Evidence for Aerosol Transfer of SARS-CoV-2–Specific Humoral Immunity
Abstract. Infectious particles can be shared through aerosols and droplets formed as the result of normal respiration. Whether Abs within the nasal/oral fluids
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以前投稿したものとは別株です。
まだ完全に開花していません。
VaT Tsar Gorohは完全開花まで5日以上かかる気がしますね。
他のセントポーリアは開花が始まって夜お花が閉じてを3日くらい繰り返して開花に至りますが
この種はその期間が長く5日?くらいかかる感じです。
しかもストークが低い位置から開花が始まり、数日かけて伸びながら開花します。
以前開花した別株のVaT Tsar Goroh
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こちらもストーク見えてますので、お花がこのままか変化するのか興味深々です。
先日メルカリで出品したのですが、ある方にお送りした苗の根です。
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一見根張りが良いと思いがちですが、実は良くない(ダメでもないですが)根です。
以前投稿したCalifornia victoryの根と比べれば一目瞭然です。
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こちらがCalifornia victoryの根です。
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Vodyanoyまた開花しそう、でも色がこれまた変わってる〜
しかも葉焼けが酷いです。
LED使わなくなって窓際のワーディアンケースに入れ
日差しが欲しくてレースのカーテンを開けていたら
直射日光がいつの間にか入るようになり、30度くらいの高温になり
かなりの苗が葉焼け高温障害に(苦笑)
ただでさえ難易度高めのセントポーリアなのにショック。
なかなか大きくなりませんし。
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今度はダブルではなくシングル、しかしパラソル咲。
色も別株は淡い紫に紫のスプラッシュ。
こちらは濃い目の紫にピンク?のパフと紫のスプラッシュ。
うーーーーん、確かにRS-vselennayaはパラソル咲なんですよね。
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転落事故翌日の様子。
セキセイインコのケージカバーにしている毛布?の横で。
マウはインコを襲わないけれど、エミルとキャスは襲うのでなかなか放鳥できません。
ごめんよ〜、ちゅんちゅん(セキセイインコの名前)
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セキセイインコのちゅんちゅん9歳(♂)です。
いつも「元気ですか?」「ちゅんちゅ〜ん、元気」と喋っています。
セントポーリアも色々ストークがついてきたり開花してきています。
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手前SM Olesyaは開きかけ。
左奥がTomorrow’s Pink Ice 、右奥がLE Mont Saint-Michel。
電子ピアノ(クラビノーバ)の上にキャス。
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California victory
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AE Playful Wolf
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Bloom Lovers Explosion
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Vodyanoy
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RS Shelif
フリフリが少ない…
海外画像では↓
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こんな感じのセントらしいです。
苗が充実しないとダメかなぁ?
因みに寒い時期はこんなふうでした↓
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いずれにせよ、フリフリがもっと欲しいですね。
葉挿ししないと、個体差かもしれないですし。
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キヨネ。
この藤色?ラベンダー?好きです。
アッシュラベンダー?アッシュがかってます。
葉挿しから。
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バンビーノ。
トレイル風、こちらのお色も素敵。
バラでいうと青龍の色、赤みの少ない爽やかな色。
猫の餌食です。。。
株分けしてもう1つはプレゼントしました。
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開花直前でギロチンとなってしまったRS Ofelia。
うーん、やっぱりspみたいです。
以前ブログに投稿したものとは別株。
親株がsp化していたのでしょうがないか〜。
今、いただいたRS Ofeliaの葉挿しをしていますので成功して欲しい〜。
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THE magnesium miracle
Dr.Carolyn Dean(カリフォルニア州医師、自然療法医師、博士)
KIDNEYS NEED MAGNESIUM
“Magnesium and Dialysis: The Neglected Cation” is a 2015 review where the authors found that magnesium requirements need to be reevaluated in the treatment of kidney disease and the use of magnesium in dialysis patients.
There was a much earlier attempt to evaluate the need for magnesium in kidney disease, in 1993. Here is the story that opened my eyes to the importance of magnesium in kidney disease, a story told to me by well-known magnesium researcher Dr. Burton Altura. Many years ago Dr. Altura asked a colleague, a kidney disease specialist, Dr. Markell, to test his kidney patients for magnesium levels. It was agreed that both ionized magnesium and serum magnesium would be tested and compared in dialysis patients.50 The results were that people with chronic kidney disease (of all varieties) had simultaneously the highest levels of serum magnesium and the lowest levels of ionized magnesium. It appeared that their magnesium was stuck in the bloodstream and not getting into their cells. It’s not reported in the study, but when these patients took a liquid magnesium, their ionized magnesium levels improved, their serum magnesium levels became normal, their symptoms were alleviated, and their kidney function tests improved.
This anecdote explains for me why doctors fear magnesium. They just measure serum magnesium, see that the levels are elevated, and assume the worst. However, they don’t test for and therefore don’t notice that ionic magnesium is low, showing that the cells remain starved for magnesium. There is not enough magnesium in ionic form to get inside the cells to do its work. Unfortunately, the definitive test for magnesium, which measures ionized magnesium, is a research tool and not available to the public.
MAGNESIUM DEFICIENCY IN END-STAGE RENAL DISEASE, VASCULAR CALCIFICATION IN KIDNEY DISEASE
Just as doctors are finding a buildup of calcium in the coronary arteries that they follow with coronary calcium scans to assess the risk of heart disease, kidney artery calcification is a sign of progressive kidney disease. A paper by Demer and Tintut in the journal Circulation discusses a complication of chronic kidney disease called vascular calcification that is causing widespread problems.
The authors acknowledge the sad fact that most people over sixty years of age have “progressively enlarging deposits of calcium mineral in their major arteries.” The calcium buildup causes stiffness of the arteries, which results in hypertension, aortic stenosis, cardiac enlargement, angina, intermittent claudication of the lower legs, and congestive heart failure. They conclude, “The severity and extent of mineralization reflect atherosclerotic plaque burden and strongly and independently predict cardiovascular morbidity and mortality.”
A 2014 study did find that magnesium minimizes the buildup of vascular calcification by directly antagonizing phosphate and also by suppressing absorption of dietary phosphate.53 The investigators suggest that this action of magnesium allows it to act as a phosphate binder, which would be very helpful in dialysis patients who suffer excess phosphate levels. They do not mention the direct effects of magnesium on calcium—to keep it dissolved in solution in the body.
Medications.
The list of medications that can cause AFib is very long, so I’m not going to include it here. You must look up the side effects of the medications you are on and see if atrial fibrillation is listed. The most bizarre one that I’ve found is flecainide, which is an antiarrhythmia drug—yet it causes fast, irregular, pounding, or racing heartbeat or pulse. I think it’s because this drug contains six fluorine atoms, making it a fluoride compound. Fluorine binds irreversibly to magnesium, making it unavailable to the body. Paradoxically, digoxin, calcium channel blockers, beta-blockers, and anti-arrhythmia drugs can all worsen heart arrhythmia. Over-the-counter cough and cold medications are stimulants that can raise your blood pressure and increase your heart rate, which can trigger AFib. Recreational drugs such as marijuana can raise your heart rate for several hours. Cocaine can also trigger an abnormal heartbeat.
Most clients who have consulted with me about their symptoms of atrial fibrillation are very distressed about their condition. Additionally, most patients have also been traumatized by their interaction with the medical community. Doctors give AFib patients no natural or alternative options; they immediately prescribe several medications and recommend cardioversion or catheter ablation of the fibrillating area in the heart. They provide no reassurance, instead assuring patients that their condition is lifelong and incurable and will only get worse with time. If a patient doesn’t seem compliant with the standard treatment for AFib, doctors scare them into taking their drugs by warning them that they otherwise risk having a stroke or heart attack.
Since doctors do not look closely at the role that magnesium plays in AFib, they miss the opportunity to give their patients a treatment that can help the heart’s electrical disharmony. If doctors do acknowledge and prescribe magnesium, it’s usually magnesium oxide, a form that’s very poorly absorbed, causing an overwhelming laxative effect and reinforcing to doctors the notion that magnesium is ineffective! On top of that, diarrhea can flush out even more magnesium, further upsetting the electrolyte balance. Doctors focus on magnesium oxide because it’s the form that has been used in the majority of magnesium studies and they assume diarrhea is a normal side effect of taking magnesium. A reader of my blog sent me the following story of how she developed her AFib but the doctors would never admit the cause.
A reader of my blog sent me the following story of how she developed her AFib but the doctors would never admit the cause. In 2014, on June 30, I developed abdominal pain, nausea, and vomiting. Within several hours the pain localized in the right lower quadrant, so I went to the hospital ER, and after several more hours I was diagnosed with acute appendicitis.
When I woke up, I was on two IV antibiotics—Flagyl and Levaquin—in addition to IV narcotic pain meds and nausea meds. I continued to have severe nausea and vomiting—now due to the narcotics, which also had me so sedated that I was barely aware of what was going on around me. After three more days of constant vomiting, I began refusing the narcotics and my symptoms improved quite quickly.
On my fourth hospital day, I developed AFib with a rapid ventricular response and was admitted to the ICU. At that point I also refused the Levaquin, although the cardiologist assured me that it had nothing to do with my arrhythmia. From reading your material, I now know Levaquin is a fluoride drug that binds magnesium!
They finally checked my electrolytes. Serum magnesium and potassium were both low—apparently they had not been checking them postop despite the fact that I had been vomiting constantly for four days!
I converted to normal sinus rhythm fairly quickly after discontinuing Levaquin and taking magnesium and potassium and a short course of the antiarrhythmia drug amiodarone.
It is truly shocking how unaware conventional medicine is of basic biochemistry and also of the dangerous side effects of the medications they prescribe. I am very fortunate that they didn’t kill me.
Doctors believe that most cases of AFib are secondary to heart disease, so the treatment is to medicate those symptoms to try to alter the course of the disease. At one time doctors described adrenal stimulation and vagus nerve relaxation as factors in AFib. But doctors no longer discuss these causes of AFib with their patients, leaving them to worry endlessly. That happened to a client of mine who experienced arrhythmia, anxiety, and shortness of breath from drinking cold water.
Doctors say that if you have AFib, you are at increased risk for heart failure, clots, and strokes. But that’s only if you already have heart disease. Most people I speak with do not have a heart problem; they have a magnesium deficiency problem.
Unfortunately, the medications that are used to treat AFib can themselves cause heart disease, disease, which may just increase the likelihood of maintaining an AFib condition. And those patients with heart disease, high blood pressure, and high cholesterol are on medications that cause more heart disease because those meds cause magnesium deficiency. That’s probably why doctors say that AFib is incurable. They don’t know that magnesium deficiency may be the cause and magnesium supplementation may be the cure for many people.
The heart has four chambers; the top two are atria and the bottom two are ventricles. What causes the atria to fibrillate? In a healthy heart, the electrical impulses in the atria are coordinated by the proper balance and interaction of several minerals that function as electrolytes: magnesium, calcium, sodium, and potassium. It seems logical that an imbalance of these minerals is the cause and balancing them is the cure. But doctors skirt around that issue—probably because they don’t even measure magnesium in a routine electrolyte panel. Just look at your most recent blood tests and you’ll see that I’m right. They test for sodium, potassium, calcium, and chloride but not magnesium.
Kidney failure.
With kidney failure there is an inability to clear magnesium from the kidneys.
マグネシウム療法の禁忌
腎不全。
腎不全では腎臓からマグネシウムを除去できない。
Even with these contraindications, there are many exceptions to the rule. Please read the section called “Kidneys Need Magnesium”,
but it has to be a picometer, stabilized ionic.
I have found that kidney patients, even those on dialysis, can safely take a picometer, stabilized ionic form of magnesium for their debilitating leg cramps and heart palpitations.
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再現性のない論文をエビデンスにしている医者や獣医は多いでしょう。
例えば近年でいえばディオバン事件などは、製薬会社がマネーを拠出し、国立大教授を中心に製薬会社に有利な論文を書かせたのですが
実際には論文にあるような効果はなかった。
和田秀樹医師が批判するように中心となった教授達はお咎めなし、そして東大の教授に就任するも、とある医療事故が(隠蔽)
医者はこの論文をエビデンスとしてディオバンを信じて患者に投薬していたでしょうね。
多くの医者や獣医は教えられたこと、エビデンスを鵜呑みにしてしまう性質がある(日本人全般に言えるが)
恣意的な論文であろうと疑わず、見抜けない可能性が高い。
なぜなら教えられたことを忠実に再現する能力は必要だが、そこには与えられた条件以外の思考力を必要としていないからだ。
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キャス(ノルウェージャン)
現在7.4kgで体重は増えていません。
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エミル(ノルウェージャン)
現在6.08kgでこちらも体重は変わりません。
ペットショップでガリガリ300g(!)しかなかったとは思えないくらい大きくなっています。
この前、2年前の去勢時に使ったキャリーに試しに入れようとしたら入りませんでした。
当時の血液検査で成長期に高くなるALPが高値ではなく、4.8kgで成長も止まったと獣医に言われましたが
縦にも横にも更に成長したようです。
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マウ(アメショー、エミルと)
マウじいさんはもうすぐ19歳!
食欲旺盛でウェットフードもペロリ。
慢性腎不全とは思えないほどの回復ぶり。
生涯で1度もワクチンやノミダニ取り薬をしたことなし。
現在体重は4.36kg。
サプリはアサイゲルマニウム、アブチャーガ、アセチルカルニチン、ラクトフェリン、イオン化マグネシウム、はちみつ(非加熱ロシア製)
これらのサプリは他のにゃんズも摂取。
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もしかすると、リポソームグルタチオンが効いたのかもしれません。
グルタチオンは普段与えていませんでした。
理由は、グルタチオンはキレート作用があって、ミネラル等を排出してしまうからです。
マウの場合、慢性腎不全でリン等の老廃物質がグルタチオンによって除去されている可能性があります。
猫は元々グルタチオン抱合が弱いとされていて、そのために薬の解毒や有害物質の解毒ができずに中毒を起こすそうです。
そういえば、だいぶ前に書いたと思いますが、ギネス記録の猫ちゃん38歳と37歳は飼い主が同じで血縁関係もないのに長寿。
アスパラガスやブロッコリーを常食していたらしいのですが、これらにはグルタチオンが豊富なんですよね。
因みに医薬品である人間用のグルタチオン錠剤(タチオン)はグルタチオン100mgにつき、添加物が350mgだそうです。。。(ある医師がXに挙げていました)
ウェットだけでなく、殆ど飲まなかった水も飲むようになっています。 Image may be NSFW. Clik here to view.
グルタチオンの効果があったにせよ、一時的な効果だとは思いますが
数日以内に亡くなると思っていたので、予想外の展開です。
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放し飼いされているらしき猫ちゃん
ペルシャ猫でしょうか?
飼い主の方曰く、日中は家に誰もいないから家に閉じ込めておくのがかわいそうとのこと。
うちは怖くて放し飼いなんてできません。。。